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verified the therapeutic effects of L-GSH supplementation in a mouse model of tuberculosis, as manifested by a reduction in MTB burden and oxidative stress levels in the lungs, as well as an increase in lung levels of IL-12, IFN-γ, IL-2, IL-17, and TNF-α, and a decrease in IL-6, IL-10, and TGF-β production (196)

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In addition, oxidation is a trigger for activation of src tyrosine kinases (Mallozzi et al., 2001) and inhibition of tyrosine phosphatases (Zipser et al., 2002), resulting in a cumulative tyrosine hyperphosphorylation of membrane target proteins

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