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This slow rate gives time to the BSO-treated cells to adapt to the progressively oxidizing environment, setting up stress response pathways aimed at increasing cell survival (Rahman et al., The survival responses to GSH depletion include the transcriptional activation of genes encoding for cell protective proteins, via the nuclear factor kappaB (NF-kappaB) (Filomeni et al., NF-kappaB is a transcription complex that in response to many insults, including oxidative stress, mediates the transactivation of key cell protective proteins (Morgan and Liu, 2011)