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Going back to our model, this could mean that the GSH binds within the zone G2, which causes a conformational change that allows the binding and the transport of the drug by zone D, which is followed by the release of GSH

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Hypoxia induces the UPR through activation of the ER stress sensor molecules activating transcription factor 6, inositol-requiring protein 1 (IRE1), and PKR-like ER kinase (PERK) (1 in Fig