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Our findings demonstrated that GSH inhibition leads to synthetic lethality in VRK2-deficient PC cells, which exhibits impaired expression of SLC7A11 at PM level

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peripherally (i.e., portal, superior mesenteric, lienal and inferior caval vein)) and arterial (i.e., abdominal aorta thrombosis, thus low-flow state caused by cardiac dysfunction or sever volume depletion [68])