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When both rapid auditory processing and tonic inhibition are impaired, some studies have reported a paradoxical decrease in central gain in certain patient subgroups, particularly those without comorbid hyperacusis (Chen et al., 2021

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Color:Navy

This activity is primarily mediated through the blockade of pro-inflammatory cytokine production and the downregulation of cyclooxygenase-2, which constitutes a key mechanism for attenuating secondary tissue damage