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In particular, the activation of nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) and mitogen-activated protein kinase (MAPK) pathways of activated B cells are activated, leading to an increase in cytokines such as tumor necrosis factor alpha (TNF-α), interleukin-1β (IL-1β), and interleukin-6 (IL-6), leading to the suppression of the PI3K-Akt-mTOR signaling pathway, which is one of the most critical anabolic pathways, and a decrease in protein synthesis (53)