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This may be useful in the sense that resistance to apoptosis is a widespread noted effect, and cells that are resistant to apoptosis remain sensitive to ferroptosis (dos Santos et al., 2023)

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This metabolic shift changes mitochondrial ratios of acetyl-CoA and nicotinamide adenine dinucleotide (NAD+)/nicotinamide adenine dinucleotide + hydrogen (NADH), positively impacting the redox state of cells [6, 9], and limiting seizure activity [10]