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Administration of Sch B significantly inhibits glycogen synthase kinase 3β (GSK3β) activation in the hippocampus and cortex neurons of triple-transgenic Alzheimer’s disease (3 × Tg AD) mice, upregulates Nrf2, GPX4, and ferroptosis suppressor protein 1 (FSP1) expression, reduces levels of MDA, Fe 2+ , ROS, and tumor necrosis factor alpha (TNF-α), thereby suppressing neuronal ferroptosis and neuroinflammation and improving learning and memory performance

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Standardized frailty indices and healthspan scoring tools are needed for routine clinical use—paralleling human instruments like the Clinical Frailty Scale or SF-36 [71]